EurekaMag.com logo
+ Site Statistics
References:
53,869,633
Abstracts:
29,686,251
+ Search Articles
+ Subscribe to Site Feeds
EurekaMag Most Shared ContentMost Shared
EurekaMag PDF Full Text ContentPDF Full Text
+ PDF Full Text
Request PDF Full TextRequest PDF Full Text
+ Follow Us
Follow on FacebookFollow on Facebook
Follow on TwitterFollow on Twitter
Follow on LinkedInFollow on LinkedIn

+ Translate

LTB4 can directly stimulate human osteoclast formation from PBMC independent of RANKL



LTB4 can directly stimulate human osteoclast formation from PBMC independent of RANKL



Artificial Cells, Blood Substitutes, and Immobilization Biotechnology 33(4): 391-403



Leukotriene B4, as a kind of 5-lipoxygenase metabolite of arachidonic acid, is known to influence osteoclast formation and bone resorption.

(PDF emailed within 0-6 h: $19.90)

Accession: 012252060

Download citation: RISBibTeXText

PMID: 16317958

DOI: 10.1080/10731190500289784



Related references

LTB4 can stimulate human osteoclast differentiation dependent of RANKL. Artificial Cells, Blood Substitutes, and Immobilization Biotechnology 38(1): 52-56, 2010

RANKL-independent human osteoclast formation with APRIL, BAFF, NGF, IGF I and IGF II. Bone 48(4): 938-944, 2011

Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL Role of IL-6 and IL-11. Journal of Bone & Mineral Research 16(Suppl 1): S381, September, 2001

Thyroid hormones and vitamine D coordinately stimulate osteoclast formation through RANKL induction. Journal of Bone & Mineral Research 17(Suppl 1): S253, September, 2002

Bacteral CpG-DNA and viral double-stranded RNA stimulate osteoclast formation through the induction of RANKL in osteoblasts. Journal of Bone & Mineral Research 17(Suppl 1): S346, September, 2002

Platelet-released supernatants stimulate formation of osteoclast-like cells through a prostaglandin/RANKL-dependent mechanism. Bone (New York) 30(5): 726-732, 2002

Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism. Bone (New York) 32(1): 1-7, January, 2003

Phosphodiesterase inhibitors stimulate osteoclast formation via TRANCE/RANKL expression in osteoblasts: possible involvement of ERK and p38 MAPK pathways. Febs Letters 579(3): 832-838, 2005

TNF-alpha and RANKL synergistically stimulate murine osteoclast differentiation from TRAF6-deficient osteoclast progenitor cells. Journal of Bone & Mineral Research 16(Suppl 1): S210, September, 2001

Activated human T cells induce osteoclast formation from peripheral monocytes via expression of osteoclast differentiation factor /RANKL/TRANCE/OPGL. Arthritis & Rheumatism 42(9 SUPPL ): S154, Sept, 1999

Lipopolysaccharide stimulates osteoclast formation via RANKL expression independent of PGE2 production. Journal of Bone & Mineral Research 17(Suppl 1): S346, September, 2002

IL-4 inhibits TNF-α-mediated osteoclast formation by inhibition of RANKL expression in TNF-α-activated stromal cells and direct inhibition of TNF-α-activated osteoclast precursors via a T-cell-independent mechanism in vivo. Bone 51(4): 771-780, 2013

RANKL-dependent and RANKL-independent mechanisms of macrophage-osteoclast differentiation in breast cancer. Breast Cancer Research and Treatment 105(1): 7-16, 2006