+ Site Statistics
+ Search Articles
+ PDF Full Text Service
How our service works
Request PDF Full Text
+ Follow Us
Follow on Facebook
Follow on Twitter
Follow on LinkedIn
+ Subscribe to Site Feeds
Most Shared
PDF Full Text
+ Translate
+ Recently Requested

The growth of plasmodium falciparum in normal and in glucose 6 phosphate dehydrogenase deficient erythrocytes treated with the thiol oxidizing agent diamide



The growth of plasmodium falciparum in normal and in glucose 6 phosphate dehydrogenase deficient erythrocytes treated with the thiol oxidizing agent diamide



Journal of Protozoology 29(3): 535




Please choose payment method:






(PDF emailed within 1 workday: $29.90)

Accession: 029357091

Download citation: RISBibTeXText


Related references

The growth of plasmodium falciparum in normal and glucose 6 phosphate dehydrogenase deficient erythrocytes treated with a favism inducing agent. Journal of Protozoology 29(2): 315, 1982

Plasmodium falciparum: thiol status and growth in normal and glucose-6-phosphate dehydrogenase deficient human erythrocytes. Experimental Parasitology 57(3): 239-247, 1984

Plasmodium falciparum infection rates in normal and enzyme deficient erythrocytes of glucose 6 phosphate dehydrogenase deficient heterozygotes. Annual Progress Report of the S E A T O (South East Asia Treaty Organization) Medical Research Laboratories : 1971, 1970

Pentosephosphate cycle in Plasmodium falciparum-parasitized normal and glucose 6-phosphate dehydrogenase-deficient erythrocytes. Annals of Tropical Medicine and Parasitology 89(2): 162, 1995

Ribose metabolism and nucleic acid synthesis in normal and glucose-6-phosphate dehydrogenase-deficient human erythrocytes infected with Plasmodium falciparum. Journal of Clinical Investigation 77(4): 1129-1135, 1986

Inhibitory effect of a fava bean component on the in vitro development of Plasmodium falciparum in normal and glucose-6-phosphate dehydrogenase deficient erythrocytes. Blood 61(3): 507-510, 1983

The effect of purified aminoaldehydes produced by polyamine oxidation on the development in vitro of Plasmodium falciparum in normal and glucose-6-phosphate-dehydrogenase-deficient erythrocytes. Biochemical Journal 236(1): 97-101, 1986

Synergistic enhancement of a copper chelator, bathocuproine disulphonate, and cysteine on in vitro growth of Plasmodium falciparum in glucose-6-phosphate dehydrogenase-deficient erythrocytes. Southeast Asian Journal of Tropical Medicine and Public Health 34(2): 301-309, 2003

Susceptibility to hydrogen peroxide of Plasmodium falciparum infecting glucose-6-phosphate dehydrogenase-deficient erythrocytes. Parasitology 99 Pt 2: 171-174, 1989

Resistance of glucose 6 phosphate dehydrogenase deficient erythrocytes to plasmodium falciparum under conditions of di amide induced oxidant stress. Journal of Cellular Biochemistry Suppl. (7 Part A): 16, 1983

Plasmodium falciparum: merozoite invasion rate in erythrocytes with beta -thalassaemia trait and glucose-6-phosphate dehydrogenase deficient genes. Myanmar Health Sciences Research Journal 5(1): 13-18, 1993

Inhibition of the intraerythrocytic development of Plasmodium falciparum in glucose-6-phosphate dehydrogenase deficient erythrocytes is enhanced by oxidants and by crisis form factor. Tropical Medicine and Parasitology 39(4): 273-276, 1988

Relative abundance of the messenger RNA of Plasmodium falciparum glucose-6-phosphate dehydrogenase in enzyme-deficient and normal Nigerian children. British Journal of Haematology 117(Suppl. 1): 2, 2002

The adenosine triphosphate content of glucose-6-phosphate dehydrogenase-deficient and normal erythrocytes, including studies of a glucose-6-phosphate dehydrogenase-deficient man with "elevated erythrocytic ATP". Journal of Laboratory and Clinical Medicine 67(5): 726-741, 1966

Glutathione stability and oxidative stress in plasmodium falciparum infection in vitro responses of normal and glucose 6 phosphate dehydrogenase deficient cells. Biochemical and Biophysical Research Communications 109(2): 355-362, 1982