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Pharmacologic concentrations of arsenic trioxide induces growth inhibition and apoptosis in malignant lymphocytes and multiple myeloma cells


Pharmacologic concentrations of arsenic trioxide induces growth inhibition and apoptosis in malignant lymphocytes and multiple myeloma cells



Blood 92(10 Suppl 1 Part 1-2): 638A



ISSN/ISBN: 0006-4971


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(PDF emailed within 1 workday: $29.90)

Accession: 032781226

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Related references

Apoptosis and growth inhibition in malignant lymphocytes after treatment with arsenic trioxide at clinically achievable concentrations. Journal of the National Cancer Institute 91(9): 772-778, 1999

RESPONSE: re: apoptosis and growth inhibition in malignant lymphocytes after treatment with arsenic trioxide at clinically achievable concentrations. Journal of the National Cancer Institute 91(19): 1691, 1999

Arsenic trioxide-mediated growth inhibition and apoptosis in multiple myeloma cells correlated with inhibition of nuclear factor -kappaB activity. Blood 98(11 Part 1): 100a-101a, 2001

Arsenic trioxide causes apoptosis, growth inhibition and increased sensitivity to chemotherapeutic agents in multiple myeloma cells through inhibition of nuclear factor -kB activity. Proceedings of the American Association for Cancer Research Annual Meeting 43: 925, 2002

Mechanism of arsenic trioxide induced growth inhibition and apoptosis in malignant cells. Proceedings of the American Association for Cancer Research Annual Meeting 39: 68-69, 1998

Malignant cells can be sensitized to undergo growth inhibition and apoptosis by arsenic trioxide through modulation of the glutathione redox system. Blood 93(1): 268-277, 1999

Arsenic trioxide-mediated growth inhibition in MC/CAR myeloma cells via cell cycle arrest in association with induction of cyclin-dependent kinase inhibitor, p21, and apoptosis. Cancer Research 60(11): 3065-3071, 2000

Arsenic trioxide induces multiple myeloma cell apoptosis via disruption of mitochondrial transmembrane potentials and activation of caspase-3. Chinese Medical Journal 114(1): 19-24, 2001

Selective apoptosis of multiple myeloma cells in primary samples induced by arsenic trioxide. Hematology 19(6): 346-351, 2014

Effect of arsenic trioxide on induction of cell cycle arrest and apoptosis in multiple myeloma cells. Proceedings of the American Association for Cancer Research Annual Meeting 40: 226, 1999

Arsenic trioxide induces apoptosis of p53 null osteosarcoma MG63 cells through the inhibition of catalase. Medical Oncology 29(2): 1328-1334, 2012

Arsenic trioxide and melarsoprol induce apoptosis in myeloma cells in vitro and inhibit plasma cells growth in vivo. British Journal of Haematology 102(1): 328, 1998

SENP1 inhibition induces apoptosis and growth arrest of multiple myeloma cells through modulation of NF-κB signaling. Biochemical and Biophysical Research Communications 460(2): 409-415, 2015

Arsenic trioxide induces apoptosis and inhibits the growth of human liver cancer cells. Life Sciences 205: 9-17, 2018

Arsenic trioxide induces the apoptosis of human breast cancer MCF-7 cells through activation of caspase-3 and inhibition of HERG channels. Experimental and Therapeutic Medicine 2(3): 481-486, 2011