+ Site Statistics
+ Search Articles
+ PDF Full Text Service
How our service works
Request PDF Full Text
+ Follow Us
Follow on Facebook
Follow on Twitter
Follow on LinkedIn
+ Subscribe to Site Feeds
Most Shared
PDF Full Text
+ Translate
+ Recently Requested

Interleukin-1 beta and tumor necrosis factor alpha stimulate the shedding of interleukin-6 receptor in osteoblastic cells

Interleukin-1 beta and tumor necrosis factor alpha stimulate the shedding of interleukin-6 receptor in osteoblastic cells

Journal of Bone & Mineral Research 15(Suppl 1): S493

Please choose payment method:

(PDF emailed within 1 workday: $29.90)

Accession: 035160904

Download citation: RISBibTeXText

Related references

Interleukin-1beta and tumor necrosis factor-alpha, but not interleukin-6, stimulate osteoprotegerin ligand gene expression in human osteoblastic cells. Bone 25(3): 255-259, 1999

Interleukin-6 receptor shedding is enhanced by interleukin-1beta and tumor necrosis factor alpha and is partially mediated by tumor necrosis factor alpha-converting enzyme in osteoblast-like cells. Arthritis and Rheumatism 52(1): 84-93, 2005

Interleukin 1 beta and tumor necrosis factor alpha stimulate the release of gonadotropin releasing hormone and interleukin 6 by primary cultured rat hypothalamic cells. Acta Endocrinologica 123(4): 476-480, 1990

Interleukin 2 il 2 stimulates the production of interleukin 1 beta il 1 beta interleukin 1 alpha il 1 alpha and tumor necrosis factor alpha tnf alpha from human peripheral blood mononuclear cells pbmc. Journal of Leukocyte Biology 42(5): 545, 1987

Analysis of tumor necrosis factor-alpha, interleukin-6, interleukin-1beta, soluble tumor necrosis factor receptors I and II, interleukin-6 soluble receptor, interleukin-1 soluble receptor type II, interleukin-1 receptor antagonist, and protein in the synovial fluid of patients with temporomandibular joint disorders. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontics 99(3): 276-284, 2005

Interleukin 1 alpha induced interleukin 1 beta and tumor necrosis factor alpha and inhibition by interleukin 1 receptor antagonist. American Review of Respiratory Disease 145(4 Part 2): A270, 1992

Cytokine responses to intraventricular injection of interleukin 2 into patients with leptomeningeal carcinomatosis rapid induction of tumor necrosis factor alpha interleukin 1 beta interleukin 6 gamma interferon and soluble interleukin 2 receptor m r 55000 protein. Cancer Research 52(5): 1123-1128, 1992

Plasma Concentrations of Interleukin-1β, Interleukin-6, Soluble Interleukin-2 Receptor and Tumor Necrosis Factor α of Depressed Patients in Japan. Neuropsychobiology 43(2): 59-62, 2001

Endotoxin, interleukin-1-beta, interleukin-6, or tumor necrosis factor-alpha do not acutely stimulate isolated murine myometrial contractile activity. American Journal of Obstetrics & Gynecology 169(6): 1424-1427, 1993

Cytokine (interleukin-1 alpha, interleukin-1 beta, tumor necrosis factor alpha, and interleukin-6)-possessing cells in lymph nodes of malignant lymphoma. Pathology Research and Practice 189(1): 18-25, 1993

Interleukin-1 binding and prostaglandin E2 synthesis by amnion cells in culture: regulation by tumor necrosis factor-alpha, transforming growth factor-beta, and interleukin-1 receptor antagonist. Biochimica et Biophysica Acta 1181(1): 31-36, 1993

Interleukin 8 modulates interleukin-1 beta, interleukin-6 and tumor necrosis factor-alpha release from normal human mononuclear cells. Immunopharmacology 27(3): 207-214, 1994

Involvement of tumor necrosis factor-alpha, interleukin-1 beta, interleukin-8, and interleukin-1 receptor antagonist in acute lung injury caused by local Shwartzman reaction. Pathology International 47(1): 16-24, 1997

Activation of human natural killer cells by lipopolysaccharide and generation of interleukin 1 alpha beta tumor necrosis factor and interleukin 6 effect of il 1 receptor antagonist. Immunology 73(4): 450-456, 1991

Neuropeptides stimulate production of interleukin-1beta, interleukin-6, and tumor necrosis factor-alpha in human dental pulp cells. Inflammation Research 53(5): 199-204, 2004