+ Site Statistics
+ Search Articles
+ PDF Full Text Service
How our service works
Request PDF Full Text
+ Follow Us
Follow on Facebook
Follow on Twitter
Follow on LinkedIn
+ Subscribe to Site Feeds
Most Shared
PDF Full Text
+ Translate
+ Recently Requested

Release of active transforming growth factor-beta1 is stimulated by angiotensin II through both PI-3 kinase and MAP kinase pathways in kidney mesangial cells



Release of active transforming growth factor-beta1 is stimulated by angiotensin II through both PI-3 kinase and MAP kinase pathways in kidney mesangial cells



Journal of the American Society of Nephrology 12(Program and Abstract Issue): 571A




Please choose payment method:






(PDF emailed within 1 workday: $29.90)

Accession: 035644718

Download citation: RISBibTeXText


Related references

Assessment of the role of the MAP Kinase and PI3 Kinase pathways in mediating growth factor effects on kidney epithelial cells through expression of a constitutively active MAP Kinase Kinase. Proceedings of the American Association for Cancer Research Annual Meeting 44: 253, 2003

Transforming growth factor-beta1 stimulates vascular endothelial growth factor 164 via mitogen-activated protein kinase kinase 3-p38alpha and p38delta mitogen-activated protein kinase-dependent pathway in murine mesangial cells. Journal of Biological Chemistry 279(32): 33213-9, 2004

The phosphatidylinositol 3-kinase/Akt pathway enhances Smad3-stimulated mesangial cell collagen I expression in response to transforming growth factor-beta1. Journal of Biological Chemistry 279(4): 2632-2639, 2003

Transforming growth factor-beta1 stimulates protein kinase A in mesangial cells. Journal of Biological Chemistry 273(14): 8522-8527, 1998

Mesangial cells stimulated by immunoglobin A1 from IgA nephropathy upregulates transforming growth factor-beta1 synthesis in podocytes via renin-angiotensin system activation. Archives of Medical Research 41(4): 255-260, 2010

Ras- and mitogen-activated protein kinase kinase-dependent and -independent pathways in p21Cip1/Waf1 induction by fibroblast growth factor-2, platelet-derived growth factor, and transforming growth factor-beta1. Cell Growth and Differentiation 10(9): 621-628, 1999

Role of the protein kinase p38 in transforming growth factor-beta1-induced collagen expression in mesangial cells. Journal of the American Society of Nephrology 9(PROGRAM AND ABSTR ISSUE): 513A, Sept, 1998

Role of p38 MAP kinase and ERK in mediating transforming growth factor-beta1 induced cyclooxygenase-2 gene expression in human mesangial cells. Molecular Biology of the Cell 13(Supplement): 154a, 2002

Transforming growth factor-beta1 stimulated protein kinase B serine-473 and focal adhesion kinase tyrosine phosphorylation dependent on cell adhesion in human hepatocellular carcinoma SMMC-7721 cells. Biochemical and Biophysical Research Communications 312(2): 388-396, 2003

Antioxidants inhibit transforming growth factor-beta1 autoinduction in human mesangial cells The role of the p38 mitogen activated protein kinase pathway. Journal of the American Society of Nephrology 12(Program and Abstract Issue): 850A, 2001

Neuroprotection by transforming growth factor-beta1 involves activation of nuclear factor-kappaB through phosphatidylinositol-3-OH kinase/Akt and mitogen-activated protein kinase-extracellular-signal regulated kinase1,2 signaling pathways. Neuroscience 123(4): 897-906, 2004

Transforming growth factor-beta1 up-regulates the expression of nerve growth factor through mitogen-activated protein kinase signaling pathways in dental pulp cells. European Journal of Oral Sciences 115(1): 57-63, 2007

Activation of extracellular signal-regulated kinase 1 and activator protein-1 by transforming growth factor-beta1 in mesangial cells is dependent on TGF-beta type II receptor. FASEB Journal 12(4): A165, March 17, 1998

Mechanical stretch-induced fibronectin and transforming growth factor-beta1 production in human mesangial cells is p38 mitogen-activated protein kinase-dependent. Diabetes 49(4): 655-661, 2000

X-linked inhibitor of apoptosis (XIAP) inhibits c-Jun N-terminal kinase 1 (JNK1) activation by transforming growth factor beta1 (TGF-beta1) through ubiquitin-mediated proteosomal degradation of the TGF-beta1-activated kinase 1 (TAK1). Journal of Biological Chemistry 280(46): 38599-38608, 2005