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Modulations of substrate entry into cells


, : Modulations of substrate entry into cells. Methods in Enzymology 39: 495-513



Accession: 040720575

PMID: 1152686

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Neuenschwander, P.F.; Deadmond, K.J.; Zepeda, K.; Rutland, J., 2013: Correlation of factor IXa subsite modulations with effects on substrate discrimination. A key feature of factor IXa is its allosteric transformation from an enzymatically latent form into a potent procoagulant. Although several small molecules have been found to be capable of partially affecting FIXa function (i.e. ethylene glycol, C...

Anonymous, 2012: Correlation of Factor IXa Subsite Modulations with Effects on Substrate Discrimination. Key feature of factor IXa (fIXa) is its allosteric transformation from an enzymatically latent form into a potent procoagulant. Whilst several small molecules have been found capable of partially effecting fIXa function (i.e. ethylene glycol, calc...

Cunane, L.M.; Chen, Z.W.; Shamala, N.; Mathews, F.S.; Cronin, C.N.; McIntire, W.S., 2000: Structures of the flavocytochrome p-cresol methylhydroxylase and its enzyme-substrate complex: gated substrate entry and proton relays support the proposed catalytic mechanism. The degradation of the toxic phenol p-cresol by Pseudomonas bacteria occurs by way of the protocatechuate metabolic pathway. The first enzyme in this pathway, p-cresol methylhydroxylase (PCMH), is a flavocytochrome c. The enzyme first catalyzes th...

Ye, X.; Wei, Y.; Nalepa, G.; Harper, J.Wade., 2003: The cyclin E/Cdk2 substrate p220(NPAT) is required for S-phase entry, histone gene expression, and Cajal body maintenance in human somatic cells. Cyclin E/Cdk2, a central regulator of the G1/S transition, coordinates multiple cell cycle events, including DNA replication, centrosome duplication, and activation of the E2F transcriptional program. Recent studies suggest a role for cyclin E/Cdk...

Chiono, M.; Mahey, R.; Tate, G.; Cooper, D.M., 1995: Capacitative Ca2+ entry exclusively inhibits cAMP synthesis in C6-2B glioma cells. Evidence that physiologically evoked Ca2+ entry regulates Ca(2+)-inhibitable adenylyl cyclase in non-excitable cells. Elevation of cytosolic free Ca-2+ inhibits the type VI adenylyl cyclase that predominates in C6-2B cells. However, it is not known whether there is any selective requirement for Ca-2+ entry or release for inhibition of cAMP accumulation to occur....

Kay R.H., 1986: Human detectability of simple non sinusoidal frequency modulations and amplitude modulations of sounds. Journal of Physiology (Cambridge) 371: 40P

Jan, C.R.; Ho, C.M.; Wu, S.N.; Tseng, C.J., 1999: Multiple effects of 1-(beta-(3-(4-methoxyphenyl)propoxy)-4-methoxy-phenethyl)-1H-imidazole hydrochloride (SKF 96365) on Ca2+ signaling in MDCK cells: Depletion of thapsigarin-sensitive Ca2+ store followed by capacitative Ca2+ entry, activation of a direct Ca2+ entry, and inhibition of thapsigargin-induced capacitative Ca2+ entry. The effect of 1-(beta-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenethyl)-1H-imidazole hydrochloride (SKF 96365) on Ca2+ signaling in Madin Darby canine kidney (MDCK) cells was examined. SKF 96365 at 25-100 muM evoked a robust (Ca2+)i transient in a...

Jan, C.R.; Ho, C.M.; Wu, S.N.; Tseng, C.J., 1999: Multiple effects of 1-[beta-[3-(4-methoxyphenyl)propoxy]-4-methoxyphenethyl]-1H-imidazole hydrochloride (SKF 96365) on Ca2+ signaling in MDCK cells: depletion of thapsigargin-sensitive Ca2+ store followed by capacitative Ca2+ entry, activation of a direct Ca2+ entry, and inhibition of thapsigargin-induced capacitative Ca2+ entry. The effect of 1-[beta-[3-(4-methoxyphenyl)propoxy]-4-methoxyphenethyl]-1H-imidazole hydrochloride (SKF 96365) on Ca2+ signaling in Madin Darby canine kidney (MDCK) cells was examined. SKF 96365 at 25-100 microM evoked a robust [Ca2+]i transient in...

Islam, M.N.; Narayanan, B.; Ochs, R.S., 2002: A direct entry model for capacitative calcium entry by Jurkat cells. We determined Ca2+ concentrations in Jurkat cells in both the endoplasmic reticulum (ER) and cytosol with fluorescent dyes. Addition of exogenous Ca2+ to Jurkat cells caused a direct increase of ER Ca2+ despite the presence of thapsigargin (Tg), w...

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